Please use this identifier to cite or link to this item: http://ri.uaemex.mx/handle20.500.11799/79793
DC FieldValueLanguage
dc.creatorGABRIEL JOSE HORTA BAAS-
dc.creatorMARIA DEL SOCORRO ROMERO FIGUEROA-
dc.creatorALVARO JOSE MONTIEL JARQUIN-
dc.creatorMARIA LUISA PIZANO ZARATE-
dc.creatorJAIME GARCIA MENA-
dc.creatorNINFA RAMIREZ DURAN-
dc.date2017-10-14-
dc.date.accessioned2022-04-21T05:17:53Z-
dc.date.available2022-04-21T05:17:53Z-
dc.identifierhttp://hdl.handle.net/20.500.11799/79793-
dc.identifier.urihttp://ri.uaemex.mx/handle20.500.11799/79793-
dc.descriptionCharacterization and understanding of gut microbiota has recently increased representing a wide research 􀄗eld, especially in autoimmune diseases. Gut microbiota is the major source of microbes which might exert bene􀄗cial as well as pathogenic effects on human health. Intestinal microbiome’s role as mediator of in􀄚ammation has only recently emerged. Microbiota has been observed to differ in subjects with early rheumatoid arthritis compared to controls, and this 􀄗nding has commanded this study as a possible autoimmune process. Studies with intestinal microbiota have shown that rheumatoid arthritis is characterized by an expansion and/or decrease of bacterial groups as compared to controls. In this review, we present evidence linking intestinal dysbiosis with the autoimmune mechanisms involved in the development of rheumatoid arthritis.-
dc.languageeng-
dc.publisherHindawi-
dc.rightsinfo:eu-repo/semantics/openAccess-
dc.rightshttp://creativecommons.org/licenses/by-nc-nd/4.0-
dc.subjectIntestinal Dysbiosis-
dc.subjectRheumatoid Arthritis-
dc.subjectGut Microbiota-
dc.subjectRheumatoid Arthritis-
dc.subjectinfo:eu-repo/classification/cti/3-
dc.titleIntestinal Dysbiosis and Rheumatoid Arthritis: A Link between Gut Microbiota and the Pathogenesis of Rheumatoid Arthritis-
dc.typearticle-
dc.audiencestudents-
dc.audienceresearchers-
item.grantfulltextnone-
item.fulltextNo Fulltext-
Appears in Collections:Producción
Show simple item record

Google ScholarTM

Check


Items in DSpace are protected by copyright, with all rights reserved, unless otherwise indicated.